- A single ataxia telangiectasia gene with a product similar to PI-3 kinase.
- Ataxia-telangiectasia: mild neurological presentation despite null ATM mutation and severe cellular phenotype.
- Genomic Organization of the ATM gene.
- Inactive Atm abrogates DSB repair in mouse cerebellum more than does Atm loss, without causing a neurological phenotype.
